Opening & Closing
Let’s get MEDICS off to a great start! The Opening Ceremony is the first in our series of inspiring events - a chance to meet and get acquainted.
WHO WILL BE THERE?
Some of our wonderful speakers and trainers will join us. Two special Opening Lectures will mark the debut of this year’s edition:
Conny Kopp-Scheinpflug | Division of Neurobiology, Ludwig Maximilians University of Munich, Germany
The Sound of Silence and Endogenous Stress-Coping
Encoding the beginning and the end of a sensory stimulus is a fundamental process in our brains. This is the molecular mechanism that enables us to see shapes, detect movements, to be aware of physical contact or to perceive vocal communication. Even if we know a lot about how a stimulus’ onset is encoded, we know a much less about the ionic offset-encoding mechanism. For instance, it is not yet understood how changes in the acoustic environment affects offset-encoding - in other words, how do we detect the sound of silence? For her Conference, Conny Kopp-Scheinpflug will reveal some of the mechanisms behind sound-processing and will present future directions of research.
A great start needs a great finish! The Closing Ceremony will be the last MEDICS event - a heartwarming ending to our 4-day scientific journey.
The MEDICS 2017 Awards Ceremony
This is when we’ll announce the winners of the MEDICS Sessions and we’ll hand out the prizes.
Daniela Popa MD PhD | Neuroscience Department, Ecole Normale Superieure de Paris
Cerebello-cortical Network and Parkinson's Disease
The cerebellum, together with the basal ganglia, constitutes a major subcortical afferent structure of the motor cortex. In Parkinson's disease, midbrain dopaminergic neuron degeneration leads to major
functional changes in the basal ganglia-thalamo-cortical pathways in patients. In contrast, alterations of the cerebellum and of the cerebello-cortical pathway have been less examined. The Lecture will review the literature on the cerebello-cortical network in Parkinson's and will present some personal results, in animal models, about the cerebellar contribution to the functional alterations observed in Parkinson's disease.